In this lesson on glycoprotein (GP) IIb/IIIa (GPIIb/IIIa) inhibitors, you’ll learn the high-yield, test-relevant concepts behind the adjunct antiplatelet drug therapy used in the acute management of patients experiencing an acute myocardial infarction (AMI; NSTEMI, STEMI) and undergoing PCI or coronary stent placement. In this lecture from This is Why, Dr. Busti will break down how platelet aggregation inhibitors work, when they’re used clinically, and the key differences between tirofiban and eptifibatide —so you can apply the “why” behind therapy (not just memorize drug names).

You’ll learn how to:

- The GPIIb/IIIa receptor and how blocking it reduces platelet activation

- Where GPIIb/IIIa inhibitors fit in clinical practice

- High-yield uses: acute coronary syndrome (ACS) and PCI/stents

- Major adverse effects (including bleeding) and top safety pearls

The goal = make medical education easy and clinically relevant.

Access bonus materials and downloads from this episode at: https://www.thisiswhy.health/topics/gpiibiiia-inhibitors-tirofiban-eptifibatide-antiplatelet-pharmacology-review

Get more with a free membership at https://www.thisiswhy.health/

- Access free downloads from our videos

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- Organize content via playlists & collections

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- Receive member newsletters

- Coupons & discounts for exam prep resources

If this helped you, please like, subscribe, and share it with a classmate or colleague. That will help this new channel continue producing free, high-yield medical education content.

Don’t forget to turn on notifications so you don’t miss upcoming lectures in pharmacology, medical rounds, and more!

#gpiibiiiainhibitors #tirofiban #antiplateletpharmacology #eptifibatide #drbusti

Disclaimer:

This content is for educational purposes only and is not medical advice. It does not replace individualized evaluation, diagnosis, or treatment. Always seek the advice of a qualified health provider with questions about a medical condition and never delay care because of educational content.

In this lesson on P2Y12 inhibitors (ADP inhibitors), you’ll learn the high-yield, test-relevant concepts behind the most common antiplatelet drugs used in cardiovascular care. In this lecture from This is Why, Dr. Busti will break down how platelet aggregation inhibitors work, when they’re used clinically, and the key differences between clopidogrel (Plavix), prasugrel (Effient), ticagrelor (Brilinta), and cangrelor (Kengreal)—so you can apply the “why” behind therapy (not just memorize drug names).

You’ll learn how to:

- The P2Y12 (ADP) receptor and how blocking it reduces platelet activation

- Where P2Y12 inhibitors fit in dual antiplatelet therapy (DAPT)

- High-yield uses: acute coronary syndrome (ACS), PCI/stents, and secondary prevention

- Major adverse effects (including bleeding) and top safety pearls

- The exam-favorite comparisons: prodrug activation, onset/offset, reversibility, and IV vs PO options

The goal = make medical education easy and clinically relevant.

Access bonus materials and downloads from this episode at: https://www.thisiswhy.health/topics/p2y12-adp-inhibitors-antiplatelet-pharmacology-review

Get more with a free membership at https://www.thisiswhy.health/

- Access free downloads from our videos

- Access deep dive content from Dr. Busti

- Organize content via playlists & collections

- Join live Q&A

- Receive member newsletters

- Coupons & discounts for exam prep resources

If this helped you, please like, subscribe, and share it with a classmate or colleague. That will help this new channel continue producing free, high-yield medical education content.

Don’t forget to turn on notifications so you don’t miss upcoming lectures in pharmacology, medical rounds, and more!

#p2y12inhibitors #clopidogrel #antiplateletpharmacology #ticagrelor #drbusti

Disclaimer:

This content is for educational purposes only and is not medical advice. It does not replace individualized evaluation, diagnosis, or treatment. Always seek the advice of a qualified health provider with questions about a medical condition and never delay care because of educational content.

Master Aspirin (ASA) fast with this Antiplatelet Pharmacology Made Easy & Relevant lecture. In this lecture from This is Why, Dr. Busti will make sure you learn how acetylsalicylic acid works as an antiplatelet drug, including COX-1 inhibition, decreased thromboxane A2 (TXA2), and reduced platelet aggregation—plus the key clinical takeaways for MI prevention, stroke prevention, and secondary prevention.

You’ll learn how to:
- Aspirin mechanism of action (COX inhibitor → ↓ TXA2 → ↓ platelet aggregation)
- Why aspirin is different from other NSAIDs in antiplatelet therapy
- High-yield uses in cardiovascular pharmacology (MI/stroke prevention)
- Side effects, contraindications, and safety pearls you need for exams

The goal = make medical education easy and clinically relevant.

👉 Access bonus materials and downloads from this episode at: https://www.thisiswhy.health/topics/aspirin-asa-salicylic-acid-antiplatelet-pharmacology-review

👉 Get more with a free membership at https://www.thisiswhy.health/
- Access free downloads from our videos
- Access deep dive content from Dr. Busti
- Organize content via playlists & collections
- Join live Q&A
- Receive member newsletters
- Coupons & discounts for exam prep resources

👍 If this helped you, please like, subscribe, and share it with a classmate or colleague. That will help this new channel continue producing free, high-yield medical education content.

🔔 Don’t forget to turn on notifications so you don’t miss upcoming lectures in pharmacology, medical rounds, and more!

#aspirin #asa #aspirinpharmacology #antiplateletdrugspharmacology #drbusti

Disclaimer:
This content is for educational purposes only and is not medical advice. It does not replace individualized evaluation, diagnosis, or treatment. Always seek the advice of a qualified health provider with questions about a medical condition and never delay care because of educational content.